Several recent studies have confirmed that herbicide residues from genetically modified organisms have been found in the human bloodstream, crossing the maternal placenta, raising significant public health concerns and illustrating the need for labeling of genetically modified products.
A 2011 Landmark study by the Department of Obstetrics and Gynaecology, at the University of Sherbrooke Hospital Centre in Quebec, Canada has revealed that 93% of mothers tested positive for the presence of pesticides associated with GM corn. Samples were taken from 30 pregnant women and 39 other women who were not pregnant. The goal of the testing was to look for pesticide residues associated with the cultivation of GM food.
The results specify that traces of Bt toxins were found in the blood of 93 per cent of the pregnant mothers.
Twenty eight out of thirty mothers tested positive and traces were also detected in eighty percent of umbilical cords. The scientists estimate that toxins are ingested as a result of eating milk, meat and eggs from farm livestock fed GM corn.The Canadian team told the scientific journal Reproductive Toxicology: ‘This is the first study to highlight the presence of pesticides associated with genetically modified foods in maternal, fetal and non-pregnant women’s blood.’ They said the Bt toxin was ‘clearly detectable and appears to cross the placenta to the fetus’.
Another study has confirmed the mountain of evidence that substantiates the toxicity of BT protein in mammals and the prevalence in the bloodstream.
Monsanto, the world wide conglomerate associated with genetically modified organisms and herbicides, has used the gene from Bacillus thuringiensis (Bt) to genetically modify certain crops to be Bt resistant. Bacillus thuringiensis produces BT-toxin. It’s a pesticide; and it breaks open the stomach of certain insects and kills them.
The new study reveals that the Bt toxins Cry1Aa, Cry1Ab, Cry1Ac or Cry2A have toxic effects in the blood of mice and that the claims by policy regulators of the presumed nontoxicity of Bt toxin to mammals, on which all regulatory approvals of Bt crops are based, is false.
The mechanism of operation of BT toxin on insects, (the target population for the pesticide), is to break holes in the gut and rupture cells. In mice in this experiment Bt toxins, caused red blood cells to rupture and is highly disconcerting as BT toxin has crossed the placenta and was found in the umbilical cord in over eighty percent of mothers tested suggesting that it crosses into the bloodstream of babies.
The study authors stated: “It has been reported that Cry toxins exert their toxicity when activated at alkaline pH of the digestive tract of susceptible larvae, and, because the physiology of the mammalian digestive system does not allow their activation, and no known specific receptors in mammalian intestinal cells have been reported, the toxicity [of] these MCAs [microbial control agents] to mammals would negligible. However, our study demonstrated that Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A induced hematotoxicity, particularly to the erythroid lineage. This finding corroborates literature that demonstrated that alkali-solubilized Bt spore-crystals caused in vitro hemolysis in cell lines of rat, mouse, sheep, horse, and human erythrocytes and suggested that the plasma membrane of susceptible cells (erythrocytes, in this case) may be the primary target for these toxins”.
” In conclusion, results showed that the Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A can cause some hematological risks to vertebrates, increasing their toxic effects with long-term exposure. Taking into account the increased risk of human and animal exposures to significant levels of these toxins, especially through diet, our results suggest that further studies are required to clarify the mechanism involved in the hematotoxicity found in mice, and to establish the toxicological risks to non-target organisms, especially mammals, before concluding that these microbiological control agents are safe for mammals.”
The toxicity of Bt proteins in mammalian cells was also the subject of an in vitro (test-tube) study. In this study, Bt toxin Cry1Ac was found to be substantially toxic to human cells, raising substantial concerns in the human population.
Source
https://www.facebook.com/pages/Foodexposed/561781190500822
Aris A, Leblanc S. 2011, Maternal and fetal exposure to pesticides associated to genetically modified foods in Eastern Townships of Quebec, Canada. Reprod Toxicol. 2011 May;31(4):528-33. Epub 2011 Feb 18.
Mesnage et al., 2012; http://onlinelibrary.wiley.com/doi/10.1002/jat.2712/abstract.
Mezzomo, B. P., et al. (2013). Hematotoxicity of Bacillus thuringiensis as spore-crystal strains Cry1Aa, Cry1Ab, Cry1Ac or Cry2Aa in Swiss albino mice. J Hematol Thromb Dis 1(1). http://dx.doi.org/10.4172/jhtd.1000104
A 2011 Landmark study by the Department of Obstetrics and Gynaecology, at the University of Sherbrooke Hospital Centre in Quebec, Canada has revealed that 93% of mothers tested positive for the presence of pesticides associated with GM corn. Samples were taken from 30 pregnant women and 39 other women who were not pregnant. The goal of the testing was to look for pesticide residues associated with the cultivation of GM food.
The results specify that traces of Bt toxins were found in the blood of 93 per cent of the pregnant mothers.
Twenty eight out of thirty mothers tested positive and traces were also detected in eighty percent of umbilical cords. The scientists estimate that toxins are ingested as a result of eating milk, meat and eggs from farm livestock fed GM corn.The Canadian team told the scientific journal Reproductive Toxicology: ‘This is the first study to highlight the presence of pesticides associated with genetically modified foods in maternal, fetal and non-pregnant women’s blood.’ They said the Bt toxin was ‘clearly detectable and appears to cross the placenta to the fetus’.
Another study has confirmed the mountain of evidence that substantiates the toxicity of BT protein in mammals and the prevalence in the bloodstream.
Monsanto, the world wide conglomerate associated with genetically modified organisms and herbicides, has used the gene from Bacillus thuringiensis (Bt) to genetically modify certain crops to be Bt resistant. Bacillus thuringiensis produces BT-toxin. It’s a pesticide; and it breaks open the stomach of certain insects and kills them.
The new study reveals that the Bt toxins Cry1Aa, Cry1Ab, Cry1Ac or Cry2A have toxic effects in the blood of mice and that the claims by policy regulators of the presumed nontoxicity of Bt toxin to mammals, on which all regulatory approvals of Bt crops are based, is false.
The mechanism of operation of BT toxin on insects, (the target population for the pesticide), is to break holes in the gut and rupture cells. In mice in this experiment Bt toxins, caused red blood cells to rupture and is highly disconcerting as BT toxin has crossed the placenta and was found in the umbilical cord in over eighty percent of mothers tested suggesting that it crosses into the bloodstream of babies.
The study authors stated: “It has been reported that Cry toxins exert their toxicity when activated at alkaline pH of the digestive tract of susceptible larvae, and, because the physiology of the mammalian digestive system does not allow their activation, and no known specific receptors in mammalian intestinal cells have been reported, the toxicity [of] these MCAs [microbial control agents] to mammals would negligible. However, our study demonstrated that Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A induced hematotoxicity, particularly to the erythroid lineage. This finding corroborates literature that demonstrated that alkali-solubilized Bt spore-crystals caused in vitro hemolysis in cell lines of rat, mouse, sheep, horse, and human erythrocytes and suggested that the plasma membrane of susceptible cells (erythrocytes, in this case) may be the primary target for these toxins”.
” In conclusion, results showed that the Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A can cause some hematological risks to vertebrates, increasing their toxic effects with long-term exposure. Taking into account the increased risk of human and animal exposures to significant levels of these toxins, especially through diet, our results suggest that further studies are required to clarify the mechanism involved in the hematotoxicity found in mice, and to establish the toxicological risks to non-target organisms, especially mammals, before concluding that these microbiological control agents are safe for mammals.”
The toxicity of Bt proteins in mammalian cells was also the subject of an in vitro (test-tube) study. In this study, Bt toxin Cry1Ac was found to be substantially toxic to human cells, raising substantial concerns in the human population.
Source
https://www.facebook.com/pages/Foodexposed/561781190500822
Aris A, Leblanc S. 2011, Maternal and fetal exposure to pesticides associated to genetically modified foods in Eastern Townships of Quebec, Canada. Reprod Toxicol. 2011 May;31(4):528-33. Epub 2011 Feb 18.
Mesnage et al., 2012; http://onlinelibrary.wiley.com/doi/10.1002/jat.2712/abstract.
Mezzomo, B. P., et al. (2013). Hematotoxicity of Bacillus thuringiensis as spore-crystal strains Cry1Aa, Cry1Ab, Cry1Ac or Cry2Aa in Swiss albino mice. J Hematol Thromb Dis 1(1). http://dx.doi.org/10.4172/jhtd.1000104
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